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Low Carb Breckenridge 2017 is open to all including the general public, health, nutrition and fitness enthusiasts, scientific community and healthcare professionals.

For healthcare professionals, this live activity, Low Carb Breckenridge 2017, with a beginning date of 02/24/2017, has been reviewed and is acceptable for up to 19.25 Prescribed credit(s) by the American Academy of Family Physicians. Physicians should claim only the credit commensurate with the extent of their participation in the activity. AAFP Prescribed credit is accepted by the American Medical Association as equivalent to AMA PRA Category 1 credit(s)™ toward the AMA Physician’s Recognition Award. When applying for the AMA PRA, Prescribed credit earned must be reported as Prescribed, not as Category 1.

Event Page: http://denversdietdoctor.com/low-carb-breckenridge-2017/
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Saturday, February 25 • 10:00am - 10:30am
Does Fat Really Burn in the Flame of Carbohydrate? (CME Approved)

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This presentation will demonstrate that in the absence of dietary carbohydrate, anumber of systems and pathways activate and perform most of the metabolicfunctions of dietary carbohydrate. Without carbohydrate, the liver converts fat toketone bodies, making them available for oxidation in multiple tissues throughout thebody, including, most importantly, the brain. A diet low in carbohydrates (orstarvation) induces the blood-brain barrier transporter for ketone bodies, whichincreases the flow of ketone bodies into the brain to substitute, energy-wise, for themissing carbohydrates. Low insulin levels as a consequence of the reduced (orabsent) carbohydrate in the diet suppresses pyruvate dehydrogenase, so thattissues other than brain partially break down the glucose they do use to pyruvate andlactate, both of which are recycled in the liver via gluconeogenesis, thus minimizingeven the extra-cerebral use of glucose. Skeletal muscle can oxidize a number ofamino acids, and when glycogen or glucose levels are low, the carbon skeletons ofthese amino acids can move into the citric acid cycle to help maintain adequateconcentrations of CA cycle intermediates, which will drive an adequate rate ofmitochondrial respiration. In which case both fat and carbohydrate will burn in anamino acid flame. Finally, fat itself can provide substrate for gluconeogenesis viatwo different pathways. First, the glycerol moiety of triglycerides, when cleaved, canmove into the gluconeogenic pathway. And, second, the acetone, deriving from thebreakdown of fat to ketone bodies, can convert to oxaloacetate, which is agluconeogenic substrate.

Speakers
avatar for Mike Eades

Mike Eades

MD
Michael R. Eades, M.D.is the author/co-author of ten books including the NY Times bestselling Protein Power. A long-time advocate of nutritional strategies for the treatment of obesity and other diseases of civilization, Dr. Eades has used the low-carb/ketogenic approach in his practice since 1984. He has written a popular blog on nutrition since 2005.


Saturday February 25, 2017 10:00am - 10:30am
1) Forrest Room